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F., Carvalho-Oliveira, R., Jnior, G

Wu G, Fang YZ, Yang S, Lupton JR, Turner ND

Thirdly, through activation of nitric oxide synthase generating NO radicals which itself has been shown to disrupt Ca 2+ handling proteins (144, 145) but also to inhibit components of the respiratory chain and promote ROS production (146, 147)

The heart is one of the organs most vulnerable to injury and dysfunction in sepsis, and sepsis-induced myocardial injury is relatively common in the ICU ( 2 S) ameliorates cardiac dysfunction in septic rats by reducing cardiomyocyte injury caused by excessive autophagy through the activation of the AMPK/mTOR pathway ( To assess the cardioprotective capacity of compound 3b-1 , we established an in vitro model of septic cardiomyopathy using lipopolysaccharide (LPS)-challenged H9c2 cardiomyocytes

Abdelraouf, K., Chang, K.-T., Yin, T., Hu, M