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Description

This study demonstrated encouraging antitumor activity in therapy-refractory mUC treated with paclitaxel plus high-dose anti-CTLA-4 and suggests immune induction may still be possible after failure to anti-PD-(L)1 therapy

Mild soreness or redness at the injection site may occur but usually subsides within hours.

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One hotspot mutation, AKT1 E17K, significantly increases the affinity of AKT for PtdIns(4,5)P2, which activates AKT1 and promotes downstream signaling through its pathological localization to the plasma membrane (106, 107)