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Although the pathogenesis of CD has not been fully elucidated, increasing evidence suggests that programmed cell death plays a key role in disease progression

Recent studies have suggested the possibility of exploiting RNAi technology in silencing male-specific genes in the tobacco plant [135]

Dysregulation of KEAP1-NRF2 activity has been extensively demonstrated in cellular and animal models of ALS and in postmortem ALS motor cortex and spinal cord samples, so dysregulation of the NRF2/ARE antioxidant and cytoprotective pathway could be a possible mechanism underlying the progressive neurodegeneration in ALS and severity of the disease

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