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Hypermethylation silences tumor suppressor genes Hypermethylation of gene promoters is one of the key mechanisms driving drug resistance, and DNMT inhibitors (azacitidine, decitabine) can reverse abnormal methylation to enhance cisplatin-induced cytotoxicity [19]

Pediatrics 102:7376

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For shoulder conditions driven primarily by inflammation, such as bursitis, acute tendinitis, or inflammatory arthropathy, KPV may provide targeted anti-inflammatory support without the tissue-weakening effects associated with corticosteroids

Targeting oncogene expression to endothelial cells induces proliferation of the myelo-erythroid lineage by repressing the Notch pathway