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Mechanisms of EGFR Resistance in Glioblastoma

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The modified HMGB1 release dynamics promote macrophage polarization toward an M1-like phenotype, rendering MCP inhibition a dual-therapeutic strategy that concurrently induces ferroptosis and activates antitumor immunity (92)

Cancers with higher GSH levels were more likely to survive vascular endothelial-induced oxidative and nitrosative stress and metastasize at different sites [60]