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doi: 10.3389/fncir.2021.816747 157 OrtolRSotos-PrietoMCarballoACabello-PlanSKoniAMustielesVet al

Studies in RGCs, specifically, have shown that exposure to extended periods of hypoxia (achieved through vascular hypoperfusion, evoked hypoxic states, or hypoxia-mimetic agents) induced ROS and oxidative stress elevation, upregulating pathways to RGC apoptosis (Tezel, 2006

Protects cells from oxidative stress caused by toxic metal exposure

In particular, glial-neuronal communication through NMDA receptors is an essential process for proper dendritic morphogenesis and establishment of synaptic connections (Rabacchi et al., 1992