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During high-fat feeding, hepatic PRPP levels decline [94], which could be a consequence of the fact that the liver undergoes higher DNA damage, apoptosis, and compensatory proliferation, resulting in higher nucleotide synthesis rates [14]

This helps minimize the micro-tears and inflammation that typically cause delayed onset muscle soreness (DOMS), allowing you to bounce back faster and maintain training consistency without the usual downtime

However, it remains unclear how BSO actually suppresses the expression of antiapoptotic proteins

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