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[1] The following bullet points and sources outline some of the critical points that need to be made in order to understand my following theory for how these underlying mechanisms can manifest into Keratosis Pilaris, and why I believe that KP can be effectively treated by stimulating IGF-I production in skin tissue

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Later experiments have found that adding high doses of copper can increase reactive oxygen species and protein carbonylation levels, decrease glutathione levels and SOD enzyme activity, leading to depolarization of mitochondrial membrane potential, the release of cytochrome c, elevated levels of active caspase-9 and caspase-3, increased levels of Bcl2-antagonist/killer 1 (Bak1) and Bcl2-associated X protein (Bax), and decreased levels of B-cell lymphoma 2 (Bcl2), ultimately resulting in cell apoptosis (134)

Its thiol side chain often participates in enzymatic reactions, and contributes to the stability of proteins by forming disulfide bonds