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Concurrently, p53 was shown to enhance ferroptosis sensitivity by suppressing SLC7A11-mediated cysteine (Cys) uptake, while its acetylation-deficient mutant (p53 3 KR) retained regulatory capacity over ferroptosis under oxidative stress ( Recent advances have uncovered novel regulatory networks in ferroptosis

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The poliovirus-induced shut-off of cellular protein synthesis persists in the presence of 3-methylquercetin, a flavonoid which blocks viral protein and RNA synthesis