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There is a growing body of evidence supporting the variable response to carotenoid intake in increasing MP density [86, 87]

In particular, mitochondrial dysfunction is a key finding in reactive oxygen species (ROS)-induced PD pathology [4,5,6,7]

The model was first fitted to the training concentrations (37.5 and 25 M doxorubicin), and then used to simulate the test concentrations (50, 12.5 and 10 M doxorubicin) as a means of blind validation, showing good accordance between the experimental and in silico outputs for all concentrations of doxorubicin

It may also have a skin-lightening effect

Modeling of TREX1-dependent autoimmune disease using human stem cells highlights L1 accumulation as a source of neuroinflammation