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Concurrent ionic concentration shifts generate the driving force for glutamate efflux, resulting in excessive synaptic glutamate accumulation [22]

Interleukin 37 mitigated airway remodeling induced by HDM by inhibiting EMT mediated by IL-24 through the ERK1/2 and signal transducer and activator of transcription 3 (STAT3) pathways (216)

Professional APC can efficiently capture the antigen and the peptide-MHC class II complexes are formed in a post-Golgi, proteolytic intracellular compartment and subsequently exported to the plasma membrane, where they are recognized by T cell receptor (TCR) (Monaco (1995) J Leuk

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