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Regulation of potassium export channels by GSH is not yet proven in LAB and the mechanism for protection is suggested to be either sacrificial action of GSH which prevents rapid fall of intracellular pH or glutathionylation of glyceraldehyde 3-phosphate dehydrogenase (GAPDH) enzyme which helps to sustain glycolysis at the required level

Chen C, Ahn EH, Kang SS, Liu X, Alam A, Ye K (2020) Gut dysbiosis contributes to amyloid pathology, associated with C/EBP/AEP signaling activation in Alzheimers disease mouse model

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Caloric restriction increases brain mitochondrial calcium retention capacity and protects against excitotoxicity