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K., Jbara A., Rabaya O., Petrova P., Daoud S., Melliti N., et al

From a mechanistic perspective, peroxisomal -oxidation dysfunction in AP would be expected to exert a dual detrimental effect: first, by impairing the shortening of fatty acids before their transfer to mitochondria, and second, by weakening peroxisomal control of hydrogen peroxide and related redox signals

Notably, a discrepancy exists between the proportion of cells undergoing classical apoptosis and the overall reduction in cell viability measured by the CCK-8 assay (Figures 1A,B, 3A,B)

Grama CN, Suryanarayana P, Patil MA, Raghu G, Balakrishna N, Ravi Kumar MNV, et al