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Myofibroblast contributes to excessive collagen production in the atrial myocardium, leading to electrical/structural changes such as increased atrial stiffness, slow conductivity, formation of reentry circuits, and increased risk of ectopic activity, promoting and sustaining AF

11K), which is in line with a reduced severity of APAP-induced hepatocyte injury in the PDI fl/fl Alb-cre mice

The intersection of genes identified by the four algorithms are considered candidate hub genes

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