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The oncogenic Kras mutation reactivates glutamine metabolism, increases the production of reduced NADPH, and restores cellular redox homeostasis through macromolecule synthesis

From the cell cycle point of view, compared with the N0 stage, the DVL3 levels in the N1 and N2 stages of CRC metastasis were higher

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From phospholipid and fatty acid defects to altered neurotransmission: Is arachidonic acid a nexus in the pathophysiology of schizophrenia