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Bmj (2011) 342:d548

doi:10.1155/2019/4874921

In BRAF V600E-mutant CRC, tumor cells often exhibit an initial response to BRAF inhibitors, but frequently develop resistance through reactivation of the MAPK pathway, commonly via KRAS amplification or MEK mutations [76]

Cisplatin (DDP) promotes lipid peroxidation, increases MDA, ROS, promotes the expression of HO-1 and NQO-1, and induces ferroptosis of lung cancer cells, and this process can be inhibited by Fer-1 ( - pathway is one of the main mechanisms of NSCLC cell resistance to cisplatin