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Malaysian Journal of Medical Sciences

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Hyperaldosteronism is associated with impaired vascular reactivity, and it has been shown that aldosterone induces a G6PD-deficient phenotype to impair endothelial function, and aldosterone antagonism or gene transfer of G6PD improves vascular reactivity [9]

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Results: GP73 and LG2m concentrations were higher in patients with liver disease than healthy controls and higher in those with cirrhosis than fibrosis in both the development and validation cohorts