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Insulin resistance results in increased estrogen levelsboth by increasing aromatase activity, resulting in the conversion of testosterone into estrogen, and by decreasing sex-hormone-binding globulin, resulting in greater amounts of free circulating estrogen.[2] Insulin resistance also causes increased production of inflammatory mediators, which likely contributes to estrogen-sensitive conditions including fibroids

DESeq2 (1.42.1) software was used for differential expression analysis between different groups, and Metascape (v3.5) was used for pathway enrichment analysis

All participants has a CD4+ T lymphocyte count greater than 100 cells/mm^3, as well as HIV-1 RNA less than 10,000 copies/mL

metabolite concentrations were calculated from peak intensities on the basis of calibration curves from metabolite standards prepared at 2, 5, and 10 mM

Human collagen studies