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In mouse models, the team introduced combined therapy including PD-L1 blocker and cyst(e) inase (a bioengineered enzyme that degrades cystine and cysteine) and such treatment can enhance the efficacy of CTL-mediated anticancer effects, supporting the idea that inducing ferroptosis could be considered a productive way for treating cancer and overcoming immunotherapy resistance (Wang et al., 2019)

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PlGF, a member of the VEGF family predominantly expressed in the placenta, is crucial for normal pregnancy

p53 can downregulate the expression of the Xc system component SLC7A11, inhibiting cystine intake and inducing ferroptosis [61]