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Because KAT6A contains multiple functional domains, it is possible that ARTHS variants may result in a truncated protein that retains acetyltransferase activity but lacks proteinprotein interaction domains that regulate this activity

Lets see the figures

Sato, Y

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As these fats are finally transported across the inner mitochondrial membrane, the mitochondria are resupplied with the substrate necessary to resume beta-oxidation and generate robust amounts of ATP