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Chronic alcohol treatment-induced GABA-A5 histone H3K4 trimethylation upregulation leads to increased GABA-A5 expression and susceptibility to alcohol addiction in the offspring of Wistar rats

coli revealed that at least some loss of ATP upon HOCl stress is due to conversion of cellular ATP to inorganic polyphosphates (polyP), which by themselves can act like ATP-independent chaperone holdases, effective in stabilizing unfolding proteins and preventing protein aggregation both in vivo and in vitro [282, 283] (Fig