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The analysis was supported by grant from the National Research Foundation of Korea grant

Inflammation (2020) 43:172941

These studies implied TMAO-mediated aggregation of unfolded or misfolded proteins may be another mechanism linking TMAO to UPR

found that although androgen deprivation therapy inhibited the expression of renal glutaminase (KGA) in the GLS1 subtype, the expression of glutaminase C (GAC) was upregulated in tumor cells, which is an androgen-independent GLS1 subtype with stronger enzymatic activity