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(Lond.) 16 , 13 (2019)

Although in both disorders the immune response is different, genetically susceptible individuals have an inappropriate mucosal immune response against their gut microbiota, mediated by an excessive activation of effector T cell subsets and/or a deficiency of Treg cells (65) (Figure 1A)

It also reduced pro-inflammatory cytokines (IL-6, TNF-, IFN-) and the expression of the p38 MAPK pathway

Furthermore, because oxygen radicals can release iron from iron-sulfur clusters, factors that promote regeneration of iron-sulfur clusters can also play a role in recovering from oxidative damage

Juvenile parkinsonism associated with heterozygous frameshift ATP13A2 gene mutation