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In contrast, the results of qRT-PCR revealed that the expression levels of key genes ( BECN1 , ATG3 , ATG4D , ATG5 , ATG12 , and SQSTM1 ) of the autophagy signal transduction pathway in HuOCSCs overexpressing ATF4 and treated with anisomycin were significantly higher than those in the control group (Figure 6B)

While HMGB1 contributes to the development of hepatic steatosis, it may also lead to the activation of Kupffer cells and the exacerbation of inflammation [227]

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