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discovered that metformin inhibits the mitochondrial respiratory chain complex to prevent the electron transport from NADH to coenzyme Q, which somewhat lowers mitochondrial basal and maximal respiration and mildly limits ATP synthesis

2A) systems have been described as being regulated to deal with these stress conditions, both being energy-consuming [7, 69, 70]

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In addition, leptin may impair endothelial function through oxidative stress by increasing the formation of ROS that reduce the bioavailability of NO and upregulate proinflammatory cascades including adhesion and chemotactic pathways in endothelial cells [72,73,74]