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The Role of APOE4 in Aggregation of Amyloid-Beta Peptides There are two pathological hallmarks in the pathogenesis of AD: (i) the formation of extracellular amyloid plaques in the brain and (ii) the formation of intracellular neurofibrillary tangles in neurons (Hardy and Selkoe 2002)

Collectively, these findings indicate that lipid peroxidation within the TME can significantly reduce the anti-tumor functions of NK cells, highlighting the need for strategies to mitigate this effect and enhance the efficacy of NK cell-based cancer therapies

As a hub target, PPARG integrates anti-inflammatory and metabolic regulatory functions, improving vascular remodeling and right ventricular hypertrophy in PAH rats (Ji et al., 2024)

The Paragon Algorithm, a next generation search engine that uses sequence temperature values and feature probabilities to identify peptides from tandem mass spectra

Glycogen accumulation in polymorphonuclear leukocytes, and other intracellular alterations that occur during inflammation