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BMC Genomics 16 , 104 (2015)

Disruption of the NKG2A:HLA-E immune checkpoint axis to enhance NK cell activation against cancer

Indeed, GSSG addition restored PhoA activity back to WT level (Fig

It also activates key longevity pathways, including sirtuins and PARPs, which are involved in DNA repair and cellular resilience (Verdin, 2015)

Taken together, these results indicate that unrestrained fork progression induced by fork reversal deficiency in HRASV12/cyclin E1-overexpressing cells is dependent on PRIMPOL and further support a role for PRIMPOL in restarting R-loop-stalled forks via the MUS81-LIG4-ELL axis