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Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathwaysparticularly those involving proline, arginine, glutamine, and tryptophandrive collagen deposition, fibroblast activation, and disease progression

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Egner PA, Groopman JD, Wang J-S et al (2006) Quantification of aflatoxin-B 1 - N 7 -guanine in human urine by high-performance liquid chromatography and isotope dilution tandem mass spectrometry 1

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Regulation of apoptosis by the redox state of cytochrome c