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Robust optimization to reduce the impact of biological effect variation from physical uncertainties in intensity-modulated proton therapy

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In summary, AS-IV impedes the advancement of DKD-related renal fibrosis and maintains highly differentiated podocyte morphology and MC stability by negatively regulating the levels of highly activated miR-21 and inhibiting the driving effect of miR-21 on the downstream Wnt/-catenin and TGF-1/Smads signaling cascades