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By reducing oxidative stress and generating inflammatory cytokines by multiple mechanisms, such drugs can likely work together to reduce the body's overall inflammatory burden

Various approaches to evoke a preconditioned phenotype has largely been unsuccessful, highlighting several logistical problems in pharmacologic approaches to cardioprotection

Inflammasomes Large cytoplasmic multiprotein complexes that sense microbial infections or danger molecules and initiate auto-proteolytic cleavage of caspase 1, and the subsequent processing and release of pro-inflammatory cytokines, namely interleukin-1 and IL-18

Flavonoids contain several hydroxyl groups in their structure

3 Increased ROS content forces cancer cells to intensify the antioxidant mechanisms that protect them from the negative effects of these oxidative stresses