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In instances of GPX4 inactivation or GSH depletion, accumulated fatty acids and hydroperoxides undergo catalysis to form lipid peroxyl radicals in the Fenton reaction mediated by iron, ultimately resulting in cellular ferroptosis

Seeking notable changes in body composition (more muscle, less fat) and accelerated recovery from injuries or training

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These metabolic adaptions are intricately connected to critical signaling pathways like mTOR, AMPK, and HIF-1, which integrate nutrient status and inflammatory signals [11, 12]