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Its oncogenic potential is mediated by multiple mechanisms, including stimulation of cell proliferation [7, 9, 10, 13, 14], inhibition of cellular senescence [2,4] inhibition of apoptosis [8,10], resistance to oxidative stress [15], regulation of cellular metabolism [16], regulation of stem cell reprogramming [17,18], differentiation [6], and cancer stem cell maintenance [10]

A Reactive, Not Proactive Model The traditional model is built to intervene only after major damage is obvious

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Further variations on these trapping agents which enable quantitation of adduct formation within the assay include radiolabelled nucleophiles ([35 S] cysteine and [14 C]KCN) as outlined in [155, 156], fluorescent variants such as dansylated glutathione [157] and peptides containing single nucleophilic amino acid targets [86]