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This so-called autocrine loop of ROS by HSCs is regarded as mechanism corresponding to the autocrine loop of TGF- which HSCs produce in response to this cytokine with an increased collagen expression in the injured liver (Itagaki et al., 2005)

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Toll-like receptor 3 agonist poly I:C reinforces the potency of cytotoxic chemotherapy via the TLR3-UNC93B1-IFN- signaling axis in paclitaxel-resistant colon cancer

Owing to the high rate of comorbidity between motor speech dysfunction and the neurological control centers of post-stroke swallowing disorders, similar pathological mechanisms between the two have been observed, and the crossover between the two has potential clinical applications