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The charged residues like glutamic acid, lysine, and the two aspartic acids enable specific interactions with cellular receptors and signaling molecules

Nature 458:762765 Galan-Cobo A, Sitthideatphaiboon P, Qu X et al (2019) LKB1 and KEAP1/NRF2 Pathways cooperatively promote metabolic reprogramming with enhanced glutamine dependence in KRAS-mutant lung adenocarcinoma

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