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As described in the previous chapter, one effective killing mechanism of HOCl may thus be the oxidative unfolding and irreversible aggregation of essential bacterial proteins [10]

Study of heavy metals pollution and vitellogenin levels in brown trout (Salmo trutta trutta) wild fish populations

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9 Typical target genes of UPR can be correlated with protein folding, ERAD, oxidative stress, autophagy, mitochondrial dysfunction, and metabolic pathways that are induced differently due to tissue differences