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Intestinal GSH redox homeostasis is maintained by de novo synthesis[32], regeneration from GSSG[33] and GSH uptake that derives from the dietary intake and mainly from the biliary output because the bile is enriched in GSH (1-2 mmol/L)[34,35]

However, a direct role of PAs in metal-chelation has been least explored

2L, the colour, homogeneity, and fluidity of DBP-Lip-Gel had barely changed, indicating that the liposomal hydrogel maintained its physical character

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10 CastellanoI.CecereF.De VendittisA.CotugnoR.ChamberyA.Di MaroA.et al (2009)