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The protective effects of the KD appear to be mediated through metabolic reprogramming that reduces substrate availability for lipogenesis and inflammatory pathways, enhanced apoptotic regulation via caspase-3 activation and p21-mediated cell cycle arrest, and modulation of the tumor microenvironment characterized by lower IL-6 and cholesterol-driven pro-tumor signaling

Notably, changes in polyamine levels were also detected in the cerebrospinal fluid (CSF) of PD patients supporting the relevance of the data obtained in the iPSC models (124)