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Primary injury after ICH is caused by direct hematoma compression, while secondary injury involves a cascade of pathophysiological events activated by components released from the hematoma [53]

In clinical trials in Cushings disease, treatment with pasireotide resulted in increases in glucose levels, suggesting collateral pancreatic effect (57)

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Its ability to facilitate angiogenesis, the formation of new blood vessels, has also raised interest in its potential applications for improving blood flow and wound healing

Your body needs time to adapt to each dose level before taking on additional receptor activation