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Excessive MDA in liver tissue will consume a large amount of antioxidative factors, such as SOD and GSH, which can protect liver from the attacks of free radicals, but once the balance is broken, SOD and GSH will be unable to protect liver against the excessively increased MDA (22,23)

Its role as the body's primary endogenous antioxidant means it works at the cellular level to support the entire antioxidant network, rather than functioning as a single-point intervention

Classically, this effect is attributed to the fact that the phosphate form of selenium, which is essential for some steps of the gene transcription for Se-GSH-Px synthesis, would be more readily available from selenite than from selenomethionine (Yasumoto et al., 1979

The numbers of lesions per mouse also differed as a function of diet, with recipients of CD and HFD showing 2.140.40 and 1.170.17 lesions, respectively