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Consistent with its mechanistic action, CMS121 modulates lipid profiles in AD mouse models, notably reducing endocannabinoid, FA, and PUFA levels, while significantly elevating neuroapoptotic-related ceramide levels (Ates et al., 2020)

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Although we initially assumed that oxygen may enhance glutamate production by activating the TCA cycle to increase the supply of 2-OG, greater air availability did not promote 2-OG production by the CgCS+CitS strain (Figs