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Elevated phospholipid hydroperoxide glutathione peroxidase (GPX4) expression modulates oxylipin formation and inhibits age-related skeletal muscle atrophy and weakness

Excessive glutamate release activates NMDARs and AMPARs, leading to Ca 2+ overload, promoting neuronal oxidative stress, inducing mitochondrial damage, and increasing the risk of cell death

Thymosin beta4 and a synthetic peptide containing its actin-binding domain promote dermal wound repair in db/db diabetic mice and in aged mice

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