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The 5-on/2-off schedule emulates natural GH pulsatility rhythms and reduces receptor desensitization during longer overall cycle windows

Arrestins bind to the phosphorylated receptor and prevent further activation of G proteins, effectively turning off the signaling pathway

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[8] The picture that emerges is of a molecule that supports both the laying-down of new matrix and the orderly turnover of old matrix a remodeling regulator rather than a one-directional growth signal, which is how Pickarts synthesis of the tissue-remodeling literature frames it