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SKU: 64593492912
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The Journal of Clinical Pharmacology

In combination, mitochondrial dysfunction and oxidative stress significantly contribute to weakened immunity, endothelial dysfunction, and increased inflammation

10.1016/j.fct.2020.111229 96 RoccoA.CompareD.AngrisaniD.Sanduzzi ZamparelliM.NardoneG

R.Tuakli-WosornuY.SkidmoreE.SilverJ

Complex mechanisms are proposed to explain the anti-estrogenic nature of AhR in breast and other cancers [132, 133] that are associated with the direct interaction with ER, resulting in the inhibition of DNA binding and disruption of coactivator/repressor recruitment [134, 135], that are associated with the anti-proliferative and pro-apoptotic effects of AhR agonist [136]