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Concurrently, ACC inhibition reduces inflammatory macrophage recruitment through blockade of chemokine receptors CCR2/CCR5 (e.g., via cenicriviroc), thereby attenuating pro-inflammatory cytokine production in pulmonary niches (Tacke and Weiskirchen, 2018)

These findings suggest that EA treatment mitigates the worsening of symptoms following TBI, potentially by inhibiting TGIF-mediated apoptosis in secondary brain injury post-TBI

Neuropharmacology 2007

10.1016/j.freeradbiomed.2022.10.004 214 ZhangS.ZhangL.LuH.YaoY.LiuX.HouJ

Neutralizing Free Radicals: By soaking up reactive oxygen species (ROS) caused by sun exposure, it prevents the initial signal that tells cells to create more pigment