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In cancer cells, increased lactate concentrations have been reported to suppress apoptosis through HIF-1-mediated upregulation of anti-apoptotic proteins, including BCL-2 and survivin, while concurrently promoting ferroptosis resistance via SCD1 upregulation (58,141)

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Figure 1 A critical element in the pathophysiology of sepsis is endothelial dysfunction, which correlates with disease severity and mortality