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Description

N-acetylcysteine normalizes neurochemical changes in the glutathione-deficient schizophrenia mouse model during development

The binding signal from the parental scFv R023 was used as reference, and clones showing binding signal higher than the parental were recovered after sorting on an S3e FACS (Bio-Rad)

2-(2,6-Dimethylphenylamino)-5,6-dihydro-4H-thiazine

CBFs seem to delay the onset of leaf senescence via regulating their target genes, such as RD29 and COR15 to control senescence-associated genes ( SAGs ) (e.g., ANAC019 and ANAC055 ) (Yang et al., 2011