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Des travaux complementaires ont documente une activation de la voie VEGFR2 (Vascular Endothelial Growth Factor Receptor 2), impliquee dans l'angiogenese, et ont decrit une upregulation des early growth response genes (Egr-1) ainsi que du facteur de transcription FAK-paxillin dans les cellules endotheliales etudiees in vitro

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