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Brain-derived neurotrophic factor ameliorates learning deficits in a rat model of Alzheimers disease induced by a1-42

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Notably, schlank, the drosophila CERS isoform, and mammalian CERS2 are capable of responding to intracellular fatty acid levels with direct binding and upregulation of Srebf expression and, conversely, downregulated expression of intracellular lipases (74, 75)