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M6 (second part of L1 and TM12) and M8 (TM16-TM17), while retaining the ability to transport GSH, were greatly impaired, mainly for M8, in their ability to transport it in a stimulated manner and consequently to sensitize cells to GSH-depleting agents

doi: 10.2165/00003088-199937010-00002 [PubMed] 12756210 Zhang W, Ramamoorthy Y, Tyndale RF, Sellers EM

Proteomic characterization of Mycobacterium tuberculosis subjected to carbon starvation

Djukanovic, R., Roche, W.R., Wilson, J:W., Besley, C.R.W., Twentyman, O.P., Howarth, P.H., Holgate, S.T

Finally we show that the intrinsic substrate specificity of Tyr kinases has remained fundamentally unchanged from worms to humans, suggesting that the fidelity between Tyr kinases and their protein substrate sequences has been maintained across hundreds of millions of years of evolution