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however, under excitotoxic conditions, excessive MCU-mediated Ca uptake induces mitochondrial permeability transition pore (mPTP) opening, uncouples oxidative phosphorylation, and elevates reactive oxygen species (ROS) production[21, 22]

On the one hand, miR-29a-3p interacts with Keap1, thereby inhibiting Keap1 activation and subsequently inducing Nrf2 protein expression

Cells employ several mechanisms to combat toxic ROS generated by abiotic stresses like oxidants

[DOI] [PubMed] [Google Scholar] 38.Side effects of a dopamine agonist therapy for Parkinson's disease: a mini-review of clinical pharmacology

T-cell proliferation and lymphocyte function depend partly on adequate intracellular glutathione